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The key endpoint indications were changes in oxidative stress markers following treatment in the NAC group vs the non-NAC group

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These microsomal membrane-bound, heme-thiolate proteins, located mainly in the liver, but also in enterocytes, kidneys, lung, and even the brain, are responsible for the oxidation, peroxidation, and reduction of several endogenous and exogenous substrates [13, 15, 16]

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