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Kraemer and colleagues developed a new hypothesis, suggesting that supplementation with l-carnitine reduced structural and biochemical muscle damage and facilitated tissue repair by protecting against carnitine deficiency in the endothelial cells, thereby ameliorating blood flow and oxygen supply [67]

A recent study showed that PUFA-enriched neurons under ferroptotic stress are more likely to trigger lipid peroxidation (LPO) and structural disruption in adjacent endothelial cells

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The final example of a small molecule drug effective against protein aggregation is squalene (ENT-01), which prevents the initial nucleation step of -synuclein aggregation by competitively binding to negatively charged phospholipids on vesicular membranes