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Moreover, protein phosphorylation catalysed by the calcium-dependent protein kinase, CPK 28, activates APX2 activity through phosphorylation at Thr59 and Thr164 (Hu et al
Moreover, their oxidized forms are regenerated by specific reduction systems (dehydroascorbate or glutathione reductase), enabling them to act as effective regulator of the cellular redox state (Foyer and Noctor, 2011)
Forsythoside A mitigates Alzheimers-like pathology by inhibiting ferroptosis-mediated neuroinflammation via Nrf2/GPX4 axis activation
[DOI] [PMC free article] [PubMed] [Google Scholar] 204.Verma S., Anderson T.J
