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The concept of CNS inflammation (classically termed neuroinflammation) is still being conceptualized in detail and commonly refers to inflammatory processes taking place in the CNS to counteract infection, eliminate cellular debris or generally protect the integrity of the CNS

After a hemorrhagic stroke, the expression of iron-handling proteins, including transferrin, ferritin, and TfR, along with both iron-importing and -exporting proteins, divalent metal transporter 1 (DMT1) and ferroportin (FPN), is increased [132,133,134]