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Niacin in patients with low HDL cholesterol levels receiving intensive statin therapy
On the other hand, p53 suppresses ROS generation in cellular lipids by competitively binding to dipeptidyl peptidase-4 (DPP4), thereby preventing NOX1 activation
Moreover, the phosphatases, i.e., phosphatase and tensin homolog (PTEN), protein tyrosine phosphatase 1B (PTP1B), and protein phosphatase 2A (PP2A), which act via dephosphorylating the molecules in the insulin signaling, are also inhibited by the physiologically produced H 2 O 2 (Lennicke and Cocheme, 2021a
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