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However, patients with HCC often develop resistance to TKIs and this resistance development is associated with increased IGFBP-1 expression, the aggregation mechanism manifested by TKI-induced hypoxia increasing IGFBP-1 expression through activation of HIF-1 and HIF-2

Toxicol Lett 156,10715

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To improve detection and refine scoring, several synonyms were used for each KE during the computational search process, and the full list is provided in Supplementary Table S3