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Genetic as well as pharmacological inhibition of Epha4 signaling was able to rescue the mutant SOD1 phenotype in zebra fish and increased survival in mouse and rat models of ALS by promoting neuromuscular re-innervation in late stages of ALS[39]

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It is understood that a permanently increased sugar level could damage the nerves, however a B12 deficiency caused by diabetes drugs is also today a proven cause (55)