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doi: 10.3390/ANTIOX9080725 83 BotterweckAAMVerhagenHGoldbohmRAKleinjansJVan Den BrandtPA

Early preclinical studies suggested GLP-1 receptor expression in thyroid C cells, forming the basis for rodent findings of C-cell hyperplasia and medullary thyroid carcinoma under chronic GLP-1RA exposure [47,48]

Pro-inflammatory cytokines stimulate corticotropin-releasing hormone (CRH) secretion and impair glucocorticoid receptor signaling, resulting in diminished negative feedback and sustained hypercortisolemia