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Mishra B, Barik A, Kunwar A et al (2008) Correlating the GPx activity of selenocystine derivatives with one-electron redox reactions

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Reducing equivalents for the reaction are provided by oxidation of a pair of cysteine residues in class II RNRs (Booker et al., NrdZ is differentially distributed amongst the mycobacteria in a pattern that is inconsistent with conventional phylogeny (Figure 9A)

110 Despite a number of studies that reported somewhat conflicting results regarding the different cardiac phenotypes in response to increased AKT activity, it has currently been recognized that short-term appropriate AKT activation leads to physiological cardiac hypertrophy, while long-term AKT activation or AKT overexpression causes pathological cardiac hypertrophy, interstitial fibrosis, and even heart failure