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This review has synthesized the growing body of evidence positioning ferroptosis, an iron-dependent form of regulated cell death driven by lipid peroxidation, as a compelling therapeutic avenue for this recalcitrant malignancy

However, it should be noted that, at present, there are no sound estimates of important bioactive phytochemicals, such as nigellimine and TQ, in traditional NS products

Ethanol, acetaldehyde, and lipopolysaccharide (LPS) activate inflammatory cascades that drive alcohol-induced liver damage [15]

So, both in vitro and in vivo experiments have demonstrated the occurrence of ferroptosis during cerebral hemorrhage