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Given that the 4 subunit can also form synaptic receptors when clustered together with x2 subunits [44], we tested whether the acute Cdk4/6 inhibition may in turn affect GABA A receptor synaptic function

3e, aML5-PEG3, aMD4-PEG11 and aMD5-PEG11)
Redox homeostasis disruption is responsible for synaptic dysfunction, neuroinflammation, and disrupted neuronal signaling, worsening the fundamental symptoms of ASD

doi: 10.1083/jcb.141.7.1539 5 GumbinerBLowenkopfTApatiraD