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4.3 GSH metabolism: a central hub in mid-late SAE The hippocampus, with its high metabolic demands and low antioxidant capacity, is uniquely vulnerable to septic injury (Yuan et al., 2020)
By inhibiting the NF-B pathway and competitively antagonizing pro-inflammatory metabolites of arachidonic acid (such as PGE and LTB), they exert anti-inflammatory and cartilage-protective effects (39, 40)
American Society of Clinical Oncology, 2012
