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doi: 10.1016/j.immuni.2021.07.005
When cyp2e1 knockout mice that lack the ability to produce the CYP2E1 enzyme were challenged with acetaminophen, they were found to be considerably less sensitive to its hepatotoxic effects than wild-type animals, indicating that this CYP is the principal enzyme responsible for the metabolic conversion of the drug acetaminophen to its active hepatotoxic metabolite NAPQI (Figure 2)
Curr Drug Metab 5:2153 Harborne JB, Williams CA (2000) Advances in flavonoid research since 1992
Inflammation in Parkinsons disease: Mechanisms and therapeutic implications
