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Free radicals, lipid peroxidation, and decreased antioxidant enzyme activity are among the most likely primary mediators in developing nephrotic syndrome [3, 4]

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Common experimental arenas include: Telomere and cellular aging models: Evaluation of telomerase activity, telomere length dynamics, replicative lifespan, and senescence markers in cell culture and animal studies

Our research has demonstrated that FOXO4-DRI can improve vascular function by alleviating endothelial cell senescence, providing a novel therapeutic approach for related diseases