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The consequent increased generation of ROS and reactive aldehydic derivatives and the downregulation of genes for antioxidant enzymes induce an altered intracellular redox status causing oxidative stress and cell death, via ATP, NAD, and -glutamyl-cysteinyl-glycine (glutathione (GSH)) depletion [4]

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While this might not establish an ergothioneine causeand-effect relationship (e.g., lower ergothioneine levels could be a consequence of brain cell loss), it underscores the potential significance of this compound

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