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Recent advances have introduced a bispecific molecule comprising a fully human monoclonal anti-glucose-dependent insulinotropic polypeptide receptor (GIPR) antibody conjugated to two GLP-1 analog peptides via optimized amino acid linkers

Impairment of complex I leads to increased ROS production and decreased ATP synthesis, contributing to neuronal energy deficits and cell death [161]

Interestingly, we observed both metabolic and immunologic differences in the lungs of untreated and treated mice even in the absence of macroscopic metastasis

Accordingly, the interesting part of this finding that is AFB1 dysregulated calcium hemostasis and increased ROS formation, leading to neurotoxic effects on astrocytes cells in vitro and in vivo (262)