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It is conceivable that this general mechanism can explain Mercury-induced oxidative stress not only through a continuous depletion of the reduced glutathione pool, but also by interfering with enzymes operating through active sites based on vicinal thiol groups or which are regulated by glutathionylation of sensing cysteine residues

and NETs, in turn, is a known trigger for coagulation and blood vessel occlusion [359], and also involve significant erythrocyte pathologies, specifically related to rheology and oxygen carrying capacity and ultimately resulting in ischemia-reperfusion injury [352]

Its enhanced receptor affinity makes it particularly potent in cell migration assays (Jones et al., 1993)

& Debrock, S