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Description
In focal ischemia, the damaging action of oxidative stress in necrosis and apoptosis has been explained in previous studies [160, 161]
These interactions modulate GS activity, influencing cellular processes such as nitrogen assimilation, carbon metabolism, and stress responses
In the Gstp/ mice, no defects in the brain have been described, but it is not clear whether neurite formation was analyzed using these mice
Clinically, patients often feel more stable, experience fewer fluctuations in energy, and show improved tolerance to ongoing treatment stress
