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Native IGF-1 is rapidly sequestered in circulation by IGF-binding proteins (IGFBPs 16), which extend its half-life but also restrict bioavailability

Although a complex network of pathways, numerous kinases, and many phosphorylation sites are involved in eNOS activity, the regulation and phosphorylation of Ser1177 is a critical requirement for this process [28]

Chen T, Hou H, Fan Y, Wang S, Chen Q, Si L, et al

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