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Treatment of platelet concentrates with the L-carnitine modulates platelets oxidative stress and platelet apoptosis due to mitochondrial reactive oxygen species reduction and reducing cytochrome C release during storage

Histological changes are similar to those observed in the Reye's syndrome, with early production of microvesicular steatosis followed by development of centri-lobular necrosis [23]

Metabolic engineering to improve provision of the precursor metabolites L-lysine and S -adenosyL-methionine (SAM) may increase de novo production of L-carnitine by the E