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Description
Clinical and experimental evidence has confirmed the role of ferroptosis in the pathological mechanisms of IBD, including increased iron deposition, glutathione (GSH) depletion, inactivation of recombinant glutathione peroxidase 4 (GPX4), and lipid peroxidation [3, 4]
Effect of age on Agaricus bisporus PHA-B stimulated insulin release and 45Ca 2+ uptake in vitro by islets of Langerhans
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Rt kh c th tr li chnh xc Glutathione dng no tt
