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We demonstrate that Ect2 activity is regulated by PKC in a DP-dependent manner in cardiac myocytes
This extended protocol adds intermediate dose steps (0.5mg, 3mg, 6mg) that do not appear in the standard clinical trial schedule
Mitochondrial dysfunction is immensely related to the production of excessive ROS, which results in damage to cellular lipids, proteins, and other biomacromolecules, finally inducing cell apoptosis or necrosis [55,135]
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