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Adequate concentrations of glutathione are required for a variety of functions, including protection of the cell from oxidative damage quenching of oxidant species, lymphocyte activation, natural killer cell activation and lymphocyte-mediated cytotoxicity [59,60]

Bridging this gap requires the identification of robust genetic and proteomic biomarkers to facilitate not only the early differential diagnosis but also to predict individual therapeutic response

However, the lack of selectivity of currently available anti-glycolytic compounds such as 2DG limits this approach [12]

Statins have been shown to attenuate neurodegeneration by decreasing neuronal death, apoptosis, microglial activation and astrogliosis [246]