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miR-122 has been well-known to play a role in liver physiology and to be suppressed in primary hepatocellular carcinoma (HCC) suggesting that miR-122 has a tumor suppressor role (Wang et al., 2012)

Recently intriguing findings have emerged that both genetic and epigenetic alterations equally contribute to ESCC with collusion between them, which provides novel theories and understandings about therapeutic effect evaluation, potential therapeutic targets and prognostic prediction in ESCC patients [18]

It also contains a functional NF1 site that acts as a repressor (Yang et al., 2002)

Yet, the full clinical potential of most neuropeptides remains unrealized as they do not readily translocate across the BBB