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These findings implicate cysteine metabolism as a critical regulator of ferroptosis susceptibility and suggest that metabolic vulnerabilities in TNBC could be exploited to enhance treatment efficacy (74)

Cruz-Silva I, Nunes VA, Rydlewski M, Gozzo AJ, Praxedes-Garcia P, Carbonel AAF, et al

doi: 10.1038/s41419-024-07244-x 20 UcarBIErikciAKosemehmetogluKOzkulCIskitABUcarGet al
