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The endothelial damage, release of TF and intravascular TF, and elevated proinflammatory cytokines trigger immunothrombosis in the pulmonary microvasculature, yielding microclots [15]
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For instance, as an increase in drug lipophilicity resulted in a proportionally higher BBB permeation rate, many drug delivery strategies were focused on an increase in lipophilicity of the CNS drugs (25)
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