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Relevant studies have shown that it is associated mainly with conditions such as inflammatory activation and immune cell infiltration, the release of profibrotic mediators, excessive deposition of the extracellular matrix (ECM), damage to intrinsic renal cells, and a reduction in the renal microvascular system [3]
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In COVID-19 ARDS, T cells are exhausted and skewed towards glycolysis, probably as a result of reduced oxygenation of the pulmonary tissue 46
