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Impact of SPM on immunocompetent cells Airway epithelial cells (ECs) are the most important target for inhaled SPM because they are the first barrier to xenobiotics, capable of releasing various mediators [13]

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Collectively, EMT emerges as a potentially reversible hub linking epithelial stress to scar formation, suggesting stage-specific combination strategies supported by single-cell profiling, lung organoids, and targeted delivery

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