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Description
N-acetylcysteine can reduce the disulfide bonds in a dose-dependent manner, 41,42 decreasing angiotensin II and increasing angiotensin 17 (a biologically active peptide exerting many opposing actions to angiotensin II), thus protecting against lung inflammation and fibrosis

Huang L, Yu LJ, Zhang X, Fan B, Wang FZ, Dai YS, et al

High levels of ROS are responsible for the recruitment of neutrophils at the site of inflammation, thus causing endothelial damage and tissue injury

These reactions mean the body is doing its job and making antibodies
