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AM-induced endothelium-dependent vasodilation is mediated by PI3K/Art/NO/cGMP pathway, the activation of cGMP-stimulated protein kinase G and/or the production of a vasodilator prostanoid (likely prostacyclin)[42,45,46], whereas AM-induced endothelium-independent vasodilation involves the opening of K + channels (calcium-activated K + channels or ATP sensitive K + channels, probably depending upon vascular beds) and the activation of cAMP-dependent protein kinase A[42,47]

introduced the concept of employing liposomes as drug delivery vehicles, marking the beginning of research into nano-formulations [5]

BioFactors 2015, 41 (4), 242 251, DOI: 10.1002/biof.1219Google ScholarThere is no corresponding record for this reference

Oddo, S