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Intriguingly, nuclear localization of beta-catenin in the matrix of Ctnnb1 mutant mice is reduced, suggesting that Wnt signaling activity in the matrix is decreased as a consequence of beta-catenin ablation in the DP

Peritubular capillary rarefaction and associated chronic hypoxia likely result from endothelial cell death under the biochemical/physical effect of matrix expansion and increased ROS production

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