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This article presents the current state of knowledge on mitochondrial damage mechanisms in HI and discusses potential therapeutic strategies, such as modulators of mitochondrial dynamics, antioxidant compounds, or inhibitors of specific mitochondrial pathways

Quantitative activation of Nrf2 by NFT, and the subsequent rise in glutathione, were similar as observed with the potent experimental Nrf2 activator diethyl maleate

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nucleatum-reprogrammed adipocytes upregulates SLC1A5 and SLC7A11 in head and neck squamous cell carcinoma (HNSCC), leading to glutathione (GSH) accumulation and cisplatin resistance