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55 , 56 Chemoprotection by N-acetylcysteine frequently results from the inactivation of primary toxicants or reactive electrophiles that arise as metabolites or lipid peroxidation products

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Under pathological conditions, microgliaas a function of different stimuliare activated to exert neuroprotective effects (M2 phenotype) or to release inflammatory factors and neurotoxic substances (M1 phenotype)
