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While sma-3 RNAi did not blunt the lifespan extension upon mrps-5 RNAi (Fig

To achieve this, there can be only two possibilities: (i) tumor cells may exhibit mechanism(s) removing malonyl-CoA but without directing it toward the fatty acyl synthase machinery, thus relieving carnitine palmitoyltransferase 1 inhibition, and/or (ii) cancers harbor a carnitine palmitoyltransferase 1 that is not sensitive to inhibition by malonyl-CoA

Our findings that metabolic re-programming can drive spontaneous activation and quiescence exit of QSCs are also supported by studies in other somatic stem cells

doi: 10.1146/annurev-pharmtox-010818-021856