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glutathione and ovarian cancer Thioredoxin Antioxidant Pathways Synergize to Drive Initiation and Progression Reactive oxygen species and ovarian
Description
Mechanistically, IMP-activated AHR stimulated the Nrf2/HO-1/GPX4 axis and enhanced ALDH3A1 expression, thereby inhibiting ferroptosis-related Fe 2+ accumulation, ROS production, and lipid peroxidation
It has also been proposed that impaired mitochondrial function acts in an inverse manner on ROS, resulting in inadequate bioenergy provision and oxidative stress
treatment [1] discontinue any offensive medications D/C acetaminophen
See theconcern with NAC connecting to glutathionefor more information on this complex metabolic process
