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Description
During global brain ischemia, Zn might become toxic and result in selective neuronal loss independently of other mechanisms of brain damage such as excitotoxicity
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Furthermore, the procedure involved invasive measures that caused pain and stress to the animal [28]
Pro-apoptotic elements, BCL2 associated X, apoptosis regulator (BAX) and BCL2 antagonist/killer 1 (BAK1), drive processes such as mitochondrial outer membrane permeabilization and the creation of the mitochondrial permeability transition pore (MPTP), thus regulating the formation of pores in the outer mitochondrial membrane [112]
FR-4000013359245
