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These agents are particularly useful as add-on therapies but may also suffer from incomplete CNS penetration and systemic side effects such as hypertension and insomnia due to their off-target activity

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Intriguingly however, using a gshA dsbA strain in a periplasmic roGFP2 re-oxidation assay revealed that exogenous GSSG did not completely restore WT oxidation rate and final oxidation state (Fig

The effects of melatonin on mitochondria may be mediated via MT1/2 receptors, resulting in decreased ROS generation, higher antioxidant capabilities, and therefore, in less neural apoptosis, activating nuclear factor erythroid 2-related factor 2, as shown in preclinical models (24,92,103,104)