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The researchers analyzed data on roughly 1,800 patients using tirzepatide and 6,200 using semaglutide
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Given that PKC and TRPM5 are downstream effectors of Gq signalling in beta cells, our next step involved determining whether the PKC inhibitor (calphostin C) and TRPM5 blocker (TPPO) suppress the triagonist-induced increase in GSIS
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