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The main mechanism responsible for the observed permanent distal sensory loss is associated with mitochondrial damage, the death of sensory neurons (i.e., nerve cell necrosis), glia activation, and neuroinflammation [21] (Table 2)
Protein measurements with the folin phenol reagent
DHA release has been shown to counteract excessive glutamate activity following mild TBI in rodents Fig
Trussardi Donna expresses modern, sophistic
