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*Correspondence: David Wasilewski, [email protected] Krasemann, [email protected] These authors have contributed equally to this work Present address: David Wasilewski, Department of Neurosurgery, CharitUniversittsmedizin Berlin, Corporate Member of Freie Universitt Berlin, Humboldt-Universitt zu Berlin, and Berlin Institute of Health, Berlin, Germany This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

SARS-CoV-2 infection induces DNA damage, through CHK1 degradation and impaired 53BP1 recruitment, and cellular senescence

Notably, glutathione transferases catalyze the detoxication of the o-quinones aminochrome, dopachrome, adrenochrome and noradrenochrome, derived from dopamine, dopa, adrenaline and noradrenaline respectively, protecting cells from the intracellular effects of these reactive compounds [105], but there appears to be no equivalent system at work extracellularly to protect the receptors studied here