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Carnitine synthesis defect and autism With the overall goal to identify exonic copy number variants in the genome of ASD patients, Celestino-Soper and coauthors[42] analyzed 3743 samples for detecting disease-causing copy number variants (CNVs) that are not detected by most techniques used in conventional research and clinical diagnosis laboratories

Afterward, increased activity of ARG2, irrespective of plasma lipid levels, was sufficient to induce inflammatory changes and atherosclerosis formation (319)

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For plants, life is not possible without these messenger molecules as they support key physiological events throughout plant life