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We have shown that effective pharmacologic inhibition of GPX4 in PTC cells with direct inhibitor RSL3 induces a robust activation of ferroptosis, increased DNA damage, impaired DNA damage repair response due to reduced p-NPM1 levels, decreased cell viability and cell migration, as well as suppressed spheroid formation and viability in vitro

The primary antioxidant mechanism involves donating an electron from the sulfhydryl (-SH) group of the cysteine molecule MAINTAINS CELLULAR HEALTH: NAC N-Acetyl-Cysteine can help to support cells all throughout the body and in many tissues

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[DOI] [PMC free article] [PubMed] [Google Scholar] 699.Kobayashi H, et al