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Mitochondrial dysfunction can also be a result of oxidative stress occurring in neurons, microglia, and astrocytes [6,26,28,30]
These Ctr1-deficient mice exhibited marked growth retardation and reduced survival rates early after birth, likely due to insufficient copper absorption and subsequent impairment of copper-dependent enzymes and proteins (18)
(19) Cystic fibrosis leads to suppressed glutathione levels and the impaired ability to produce more at a normal rate
