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Approximately 10% of ALS patients are hereditary, called familial ALS (fALS), while the remaining 90% (sALS) occur sporadically without apparent genetic predisposition (Van Daele et al., 2023)

Hence, the crosstalk between the ER stress and ferroptosis pathways, as well as the coregulator ATF4, may be a potential target for GBM treatment [104]

Morgan B, Ezerina D, Amoako TNE, Riemer J, Seedorf M, Dick TP (2012) Multiple glutathione disulfide removal pathways mediate cytosolic redox homeostasis

moreover, homozygous individuals for GSTP1 105Ile allele, that encode the oxidative stress regulatory enzyme glutathione S-transferase pi 1, tend to suffer this type of NP more frequently than other people (Han and Smith, 2013)